lenticrisprv2 puro vector (Addgene inc)
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Addgene inc
lenticrisprv2 puro vector
Lenticrisprv2 Puro Vector, supplied by Addgene inc, used in various techniques. Bioz Stars score: 98/100, based on 517 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/lenticrisprv2/lentiCRISPRv2+puro+(Plasmid+%2398290)/pmc13007490-34-8-10
Average 98 stars, based on 517 article reviews
Lenticrisprv2 Puro Vector, supplied by Addgene inc, used in various techniques. Bioz Stars score: 98/100, based on 517 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/lenticrisprv2/lentiCRISPRv2+puro+(Plasmid+%2398290)/pmc13007490-34-8-10
Average 98 stars, based on 517 article reviews
lenticrisprv2 puro vector - by Bioz Stars,
2026-09
98/100 stars
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Clone Assay:Article Title: HIV-1 Vpr is targeted for degradation by autophagy. Article Snippet: .. For this, the guide RNAs (gRNAs) that appear below were cloned into Article Title: HER2 heterogeneous breast cancer models reveal novel therapeutic targets and subclonal dynamics during evolution to resistance to HER2-targeted therapies Article Snippet: .. Guides were cloned in Article Title: CD59 promotes pancreatic cancer progression via a tumor cell-intrinsic JAK2-STAT3 signaling axis. Article Snippet: Pancreatic cancer is a highly aggressive malignancy with limited therapeutic options.. Although the complement system has been implicated in tumor biology, its tumor cell–intrinsic roles in pancreatic cancer remain unclear.. Here, we identify the complement regulatory protein CD59 as a critical driver of pancreatic cancer progression through a cell-intrinsic signaling mechanism. Plasmid Preparation:Article Title: HIV-1 Vpr is targeted for degradation by autophagy. Article Snippet: .. For this, the guide RNAs (gRNAs) that appear below were cloned into Article Title: Microbial sensing through the non-canonical inflammasome modulates airway type 2 immunity Article Snippet: .. Cloning:Article Title: METTL16 promotes taxane resistance in Triple-Negative Breast Cancer through m 6 A-dependent translational upregulation of ABCB1 Article Snippet: PCR products were cloned into the XhoI/XbaI sites of pENTR4-FLAG (Addgene # 17423), in frame with the FLAG tag (N-terminus). .. Gateway recombinational cloning was performed to transfer FLAG-tagged WT or N184A METTL16 to the pLenti-CMV-puro destination vector (Addgene # 17452). shRNAs against human METTL16 (TRCN0000134067, TRCN0000136815, and TRCN0000136881) were obtained from Sigma-Aldrich (in pLKO.1 vector). Expressing:Article Title: METTL16 promotes taxane resistance in Triple-Negative Breast Cancer through m 6 A-dependent translational upregulation of ABCB1 Article Snippet: PCR products were cloned into the XhoI/XbaI sites of pENTR4-FLAG (Addgene # 17423), in frame with the FLAG tag (N-terminus). .. Gateway recombinational cloning was performed to transfer FLAG-tagged WT or N184A METTL16 to the pLenti-CMV-puro destination vector (Addgene # 17452). shRNAs against human METTL16 (TRCN0000134067, TRCN0000136815, and TRCN0000136881) were obtained from Sigma-Aldrich (in pLKO.1 vector). Purification:Article Title: Microbial sensing through the non-canonical inflammasome modulates airway type 2 immunity Article Snippet: .. Knock-Out:Article Title: CD59 promotes pancreatic cancer progression via a tumor cell-intrinsic JAK2-STAT3 signaling axis. Article Snippet: Pancreatic cancer is a highly aggressive malignancy with limited therapeutic options.. Although the complement system has been implicated in tumor biology, its tumor cell–intrinsic roles in pancreatic cancer remain unclear.. Here, we identify the complement regulatory protein CD59 as a critical driver of pancreatic cancer progression through a cell-intrinsic signaling mechanism. Generated:Article Title: CD59 promotes pancreatic cancer progression via a tumor cell-intrinsic JAK2-STAT3 signaling axis. Article Snippet: Pancreatic cancer is a highly aggressive malignancy with limited therapeutic options.. Although the complement system has been implicated in tumor biology, its tumor cell–intrinsic roles in pancreatic cancer remain unclear.. Here, we identify the complement regulatory protein CD59 as a critical driver of pancreatic cancer progression through a cell-intrinsic signaling mechanism. |